| PATHOLOGY: PRIMARY VIVA TABLE | |||
| Normal cell | |||
| Acute Ischaemia The initial cellular changes in acute ischaemia - | What are the biochemical hallmarks of cell injury | ||
| Describe cellular changes associated with reversible ischaemia – Describe the reversible cellular changes occurring in acute ischaemia - | Features and mechanisms of apoptosis | ||
| Describe the cellular changes associated with irreversible ischaemia - | Coagulative versus liquefactive necrosis - | ||
| Discuss the differences between reversible and irreversible cellular injury following acute ischaemia - | |||
| Disease of immunity | |||
| Type I hypersensitivity –What chemical mediators are involved | HIV - Discuss the HIV virus | ||
| Type II hypersensitivity | T Lymphocytes - | ||
| Type III hypersensitivity | Role of B cells in immune processes - | ||
| Type IV hypersensitivity | Role of complement in immune processes - | ||
| Cell injury and adaptation /Acute and chronic inflammation /Tissue repair and wound healing | |||
| Hyperplasia vs Hypertrophy [2007.1] | Pathological calcification [2007.1] | ||
| Neoplasia | |||
| Infectious Disease | |||
| Environmental Pathology | |||
| Hemopoietic system | |||
| Cardiovascular | |||
| Respiratory | |||
| Endocrine | |||
| Liver and biliary tract and Pancreas | |||
| Cirrhosis - Basic pathological principles - Pathophysiology of consequences | Hepatitis A | ||
| Acute Pancreatitis - Pathogenesis - Pathologic consequences | Hepatitis B - Serum markers in hepatitis B – Pathological consequences | ||
| Cholelithiasis - Pathogenesis and consequences | Hepatitis C - Pathogenesis and consequences | ||
| Chronic viral hepatitis - Pathogenesis | Hepatitis D | ||
| Renal system and calculi | |||
| Hemodynamic disorders, thrombosis and shock | |||
About Mike Cadogan
