Pacemaker Rhythms – Normal Patterns

Pacemaker Components

Pacemakers consist of:

1. Pulse generator

  • Power source
  • Battery
  • Control circuitry
  • Transmitter / Receiver
  • Reed Switch (Magnet activated switch)

2. Lead(s)

  • Single or multiple
  • Unipolar or bipolar

 

Pacemaker Classification

  • Pacemakers are classified by the nature of their pacing mode.
  • Classification follows pacemaker code developed by the North American Society of Pacing and Electrophysiology (NASPE) and the British Pacing and Electrophysiology Group (BPEG).
  • The NASPE/BPEG Generic (NBG) Pacemaker Code was last revised in 2002, although many textbooks still use the previous version from 1987.
  • The code is expressed as a series of up to five letters.

NBG Pacemaker Code (2002)

PositionIIIIIIIVV
Chamber(s)PacedChamber(s)SensedResponse to SensingRate ModulationMultisite Pacing
O = NoneO = NoneO = NoneO = NoneO = None
A = AtriumA = AtriumT = TriggeredR = Rate ModulationA = Atrium
V = ventricleV = ventricleI = InhibitedV = ventricle
D = Dual (A+V)D = Dual (A+V)D = Dual (T+I)D = Dual (A+V)

Position I: Chambers Paced

  • Refers to chambers paced.

Position II: Chambers Sensed

  • Refers to the location where the pacemaker senses native cardiac electrical activity.

Position III: Response to Sensing

  • Refers to pacemakers response to sensed native cardiac activity.
  • T = Sensed activity results in triggering of paced activity
  • I =  Sensed activity results in inhibition of pacing activity

Position IV: Rate Modulation

  • Indicates ability for rate modulation designed to altered heart appropriately to meet physiological needs e.g. physical activity. Sensors may measure and respond to variables including vibration, respiration, or acid-base status.

Position V: Multisite Pacing

  • Allows indication of multiple stimulation sites within one anatomical area e.g. more than one pacing site within the atria or biatrial pacing

NBG Pacemaker Code (1987)

PositionIIIIIIIVV
Chamber(s)PacedChamber(s)SensedResponse to SensingProgrammabilityAntitachydysrhythmia Functions
O = NoneO = NoneO = NoneO = NoneO = None
A = AtriumA = AtriumT = TriggeredP = SimpleP = Pacing
V = ventricleV = ventricleI = InhibitiedM = MultiprogrammableS = Shock
D = Dual (A+V)D = Dual (A+V)D = Dual (T+I)C = CommunicatingD = Dual (P+S)
R = Rate modulating

Positions I – III

  • Same as 2002 guidelines.

Position IV: Programmability / Rate Modulation

  • Reflects the programming options available for pacemaker set-up or presence of rate modulation ability.

Position V:

  • Refers to presences of anti-tachydysrhythmia functions. Superseded by the NASPE/BPEG Defibrillator Code.

 

Common Pacing Modes

AAI

  • Atrial pacing and sensing.
  • If native atrial activity sensed then pacing is inhibited.
  • If no native activity sensed for pre-determined time then atrial pacing initiated.
  • Used in sinus node dysfunction with intact AV conduction.
  • Also termed atrial demand mode.

VVI

  • Ventricle pacing and sensing.
  • Similar to AAI mode but involving ventricles instead of the atrium.
  • Used in patients with chronic atrial impairment e.g. atrial fibrillation or flutter.

DDD

  • Capable of pacing and sensing both atrium and ventricles.
  • Commonest pacing mode.
  • Atrial pacing occurs if no native atrial activity for set time.
  • Ventricular pacing occurs if no native ventricle activity for set time following atrial activity.
  • Atrial channel function is suspend during a fixed periods following atrial and ventricular activity to prevent sensing ventricular activity or retrograde p waves as native atrial activity.

Magnet mode

  • Applying a magnet to a pacemaker will initiate the magnet mode.
  • This mode varies with pacemaker set-up and manufacturer.
  • Usually initiates an asynchronous pacing mode – AOO, VOO, or DOO.
  • Asynchronous modes deliver constant rate paced stimuli regardless of native rate of rhythm.
  • In asynchronous ventricle pacing there is a risk of pacemaker-induced ventricular tachycardia.
  • Note this differs from magnet application to an Implantable Cardioversion Defibrillator (ICD) which results in defibrillator deactivation.

 

Criteria for Pacemaker Insertion

The 2002 American College of Cardiology, American Heart Association and North American Society for Pacing and Electrophysiology guidelines for implantation of cardiac pacemakers can be found here.

 

Paced ECG – Electrocardiographic Features

The appearance of the ECG in a paced patient is dependent on the pacing mode used, placement of pacing leads, device pacing thresholds, and the presence of native electrical activity.

Features of the paced ECG are:

Pacing spikes

  • Vertical spikes of short duration, usually 2 ms.
  • May be difficult to see in all leads.
  • Amplitude depends on position and type of lead.
  • Bipolar leads result in a much smaller pacing spike than unipolar leads.
  • Epicardially placed leads result in smaller pacing spikes than endocardially placed leads.

Atrial Pacing

  • Pacing spike precedes the p wave.
  • Morphology of p wave dependent of lead placement but may appear normal.

Ventricular Pacing

  • Pacing spike precedes the QRS complex.
  • Right ventricle pacing lead placement results in a QRS morphology similar to LBBB.
  • Left epicardial pacing lead placement results in a QRS morphology similar to RBBB.
  • ST segments and T waves should be discordant with the QRS complex i.e. the major terminal portion of the QRS complex is located on the opposite side of the baseline from the ST segment and T wave.

Dual Chamber Pacing

  • Dependent on areas begin paced.
  • May exhibit features of atrial pacing, ventricular pacing or both.
  • Pacing spikes may precede only p wave, only QRS complex, or both.

The absence of paced complexes does not always mean pacemaker failure as it may reflect satisfactory native conduction. ECG Features

Atrial and ventricular pacing spikes

Appropriate discordance in a ventricular paced rhythm

 

ECG Examples

Dual Chamber Pacing

Example 1

A-V sequential pacing:

  • Atrial and ventricular pacing spikes are visible before each QRS complex.
  • There is 100% atrial capture — small P waves are seen following each atrial pacing spike.
  • There is 100% ventricular capture — a QRS complex follows each ventricular pacing spike.
  • QRS complexes are broad with a LBBB morphology, indicating the presence of a ventricular pacing electrode in the right ventricle.

 

Example 2

  • A-V sequential pacing — atrial and ventricular pacing spikes precede each QRS complex with 100% capture.

 

Example 3

  •  Another example of A-V sequential pacing.

 

Ventricular Pacing

Example 4

Ventricular paced rhythm:

  • Ventricular pacing spikes precede each QRS complex (except perhaps complex #2 — although the QRS morphology in this complex is identical to the rest of the ECG, suggesting that this beat is also paced)
  • No atrial pacing spikes are seen.
  • The underlying native rhythm is probably coarse atrial fibrillation — there are several possible P waves visible in V1 but otherwise the atrial activity is chaotic.

 

Example 5

Ventricular paced rhythm:

  • Ventricular pacing spikes precede most of the QRS complexes.
  • The 6th and 7th beats are narrower, with a different morphology — these are non-paced (“capture”) beats, probably supraventricular in origin.
  • There is a pacing spike superimposed on beat #6, but this does not appear to alter its morphology — i.e. no evidence of a fusion complex.

P = paced beat, C = capture beat

 

Example 6

Ventricular paced rhythm:

  • Ventricular pacing spikes precede the QRS complexes, most of which exhibit LBBB morphology consistent with a RV pacing electrode.
  • The 5th, 6th and 11th complexes are narrower with a different morphology  — these are fusion beats produced when the ventricle is simultaneously activated by both paced and supraventricular (native) impulses. You can see how the pacing spike is shortened and the QRS duration narrowed by the co-incident native impulse.
  • The 4th complex is probably a supraventricular capture beat, although there may still be some hybridisation with a pacing spike.

P = paced beat, F = fusion beat, C = capture beat

 

Atrial Pacing

Atrially paced patients often have evidence of 1st degree AV block or Wenckebach conduction on their paced ECG that is not apparent on their baseline tracing. This is because the sort of patients that require atrial pacing (e.g. post-op cardiac surgery) commonly have some degree of AV node dysfunction (e.g. due to age-related AV-nodal degeneration / their underlying cardiac condition / post-operative ischaemia / AV-nodal blocking drugs). When these patients are paced at a faster rate than their AV node can handle, the AV node becomes “fatigued” resulting in 1st degree AV block or Wenckebach phenomenon on the paced ECG. This abnormality is not clinically important provided that the patient’s cardiac output is not compromised.

Example 7

Atrial paced rhythm with 1st degree AV block:

  • There are regular pacing spikes at 90 bpm.
  • Each pacing spike is followed by a P wave, indicating 100% atrial capture.
  • P waves are conducted to the ventricles with a prolonged PR interval (280 ms).

 

Example 8 

Atrial paced rhythm with Wenckebach conduction:

  • There are regular atrial pacing spikes at 90 bpm; each one is followed by a small P wave indicating 100% atrial capture.
  • However, not every P wave results in a QRS complex — the PR interval progressively lengthens, culminating in failure of AV conduction (“dropped QRS complexes”).
  • There is a co-existent 2nd degree AV block with Mobitz I conduction (Wenckebach phenomenon).
  • The Mobitz I is due to the patient being paced at a rate faster than his AV node can handle — at his own intrinsic rate of 50-60 bpm he had only a 1st degree AV block.

To find out the story behind this ECG, check out ECG Exigency 011

Pacemaker Puzzler

Can you work out what is going on in this ECG?

  • There is an irregularly irregular rhythm with multiple atrial and ventricular pacing spikes.
  • The majority of the QRS complexes are broad (120ms) and preceded by ventricular pacing spikes.
  • The LBBB morphology is consistent with a ventricular pacing electrode located in the right ventricle.
The varying relationship between the atrial and ventricular pacing spikes is best understood by examining the lead II rhythm strip (below):

  • Beat 1 is narrow — this appears to be a native ventricular complex triggered by an atrial pacing spike. This indicates that AV conduction is  intact to some degree (i.e. 3rd degree AV block cannot be present).
  • Beat 9 is broad with a completely different morphology and axis to the rest of the strip — this is a ventricular ectopic beat.
  • Beats 3, 6, 8, 10 and 12 are preceded by both atrial and ventricular pacing spikes — sequential A-V pacing.
  • Beats 2, 4, 5, 7 and 11 are preceded by ventricular pacing spikes only. The absence of atrial pacing spikes suggests that the pacemaker is responding to native supraventriclar impulses.
  • Given the absence of discernible P waves and the very irregular rhythm, it is likely that the underlying native rhythm is atrial fibrillation.

A = atrial pacing spike, V = ventricular pacing spike, E = ventricular ectopic beat

 

Conclusion
  • Atrial fibrillation with DDD pacing and occasional ventricular ectopics.

 

Many thanks to Dr Harry Patterson for providing us with this great ECG!

Related Topics

Further Reading

Author Credits

References

  • Brady WJ, Truwit JD. Critical Decisions in Emergency & Acute Care Electrocardiography. Wiley Blackwell 2009.
  • Bernstein AD, Daubert JC, Fletcher RD, Hayes DL, Lüderitz B, Reynolds DW et al. The revised NASPE/BPEG generic code for antibradycardia, adaptive-rate, and multisite pacing. North American Society of Pacing and Electrophysiology/British Pacing and Electrophysiology Group. Pacing Clin Electrophysiol. 2002 Feb;25(2):260-4. PMID: 11916002
  • Bernstein AD, Camm AJ, Fletcher RD, Gold RD, Rickards AF, Smyth NP, Spielman SR, Sutton R. The NASPE/BPEG generic pacemaker code for antibradyarrhythmia and adaptive-rate pacing and antitachyarrhythmia devices. Pacing Clin Electrophysiol. 1987 Jul;10(4 Pt 1):794-9. PMID: 2441363
  • Bernstein AD, Daubert JC, Fletcher RD, Hayes DL, Lüderitz B, Reynolds DW et al. The revised NASPE/BPEG generic code for antibradycardia, adaptive-rate, and multisite pacing. North American Society of Pacing and Electrophysiology/British Pacing and Electrophysiology Group. Pacing Clin Electrophysiol. 2002 Feb;25(2):260-4. PMID: 11916002
  • Chan TC, Brady WJ, Harrigan RA, Ornato JP, Rosen P. ECG in Emergency Medicine and Acute Care. Elsevier Mosby 2005.
  • Gregoratos G, Abrams J, Epstein AE, Freedman RA, Hayes DL, Hlatky MA, et al; American College of Cardiology/American Heart Association Task Force on Practice Guidelines/North American Society for Pacing and Electrophysiology Committee to Update the 1998 Pacemaker Guidelines. ACC/AHA/NASPE 2002 guideline update for implantation of cardiac pacemakers and antiarrhythmia devices: summary article: a report of the American College of Cardiology/American Heart Association Task Force on Practice Guidelines (ACC/AHA/NASPE Committee to Update the 1998 Pacemaker Guidelines). Circulation. 2002 Oct 15;106(16):2145-61.PMID: 12379588Full Text
  • Surawicz B, Knilans T. Chou’s Electrocardiography in Clinical Practice (6th edition), Saunders 2008.
  • Wagner, GS. Marriott’s Practical Electrocardiography (11th edition), Lippincott Williams & Wilkins 2007.
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